HCT-116人結(jié)腸癌細(xì)胞（附
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- 公司名稱 上海富雨生物科技有限公司
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- 更新時間 2025/2/12 8:56:31
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人結(jié)腸癌細(xì)胞HCT-116
diklerent tumor microenvironments (TME). This means that the eklicacy of immunotherapy in actual applications will, therefore, have diklerent results. Existing tumor immune subtype studies have mostly focused on immune cells, stromal cells, genes and molecules without considering the presence of microbes. Some studies have shown that
accelerated approach to introducing these therapies into the clinic to treat patients with esophageal cancer.As the first compartment of the protein secretory pathway, the endoplasmic reticulum (ER) acts as a protein synthesis factory, maintaining proteostasis and ER homeostasis. However, a variety of intrinsic and extrinsic perturbations, such as cancer, can disrupt the
| 種屬 | 人 |
| 別稱 | HCT-116; HCT.116; HCT_116; HCT116; CoCL2 |
| 組織來源 | 結(jié)腸 |
| 疾病 | 結(jié)直腸癌 |
| 傳代比例/細(xì)胞消化 | 1：2-1:3傳代 ，消化1-2分鐘 ， |
| 培養(yǎng)基配置 | McCoy’s 5A 培養(yǎng)基；10%胎牛血清；1%雙抗 |
| 簡介 | HCT116是1979年M.Brattain等從患結(jié)腸癌的男性病人中分離的三株惡性細(xì)胞之一。在半固體瓊脂糖培養(yǎng)基中形成克 隆。HCT116在無胸腺的裸鼠有致瘤性 ，形成上皮樣的腫瘤 |
| 形態(tài) | 上皮細(xì)胞樣 |
| 生長特征 | 貼壁生長 |
| 倍增時間 | ~48h |
| 基因表達(dá) | carcinoembryonic antigen (CEA) 1 ng per 1×10^6 cells per 10 days |
| 致瘤性 | Yes, in nude mice |
| STR | Amelogenin：X ，Y；CSF1PO：7 ，10；D13S317：10 ，12；D16S539：11 ，13；D18S51：16 ，17； D19S433：12 ，13；D21S11：29 ，30；D2S1338：16；D3S1358：12 ，18 ，19；D5S818：10 ，11； D7S820：11 ，12；D8S1179：12 ，14；FGA：18 ，23；TH01：8 ，9；TPOX：8；vWA：17 ，22； |
| 保藏機構(gòu) | ATCC; CCL-247 |
diklerent tumor microenvironments (TME). This means that the eklicacy of immunotherapy in actual applications will, therefore, have diklerent results. Existing tumor immune subtype studies have mostly focused on immune cells, stromal cells, genes and molecules without considering the presence of microbes. Some studies have shown that
accelerated approach to introducing these therapies into the clinic to treat patients with esophageal cancer.As the first compartment of the protein secretory pathway, the endoplasmic reticulum (ER) acts as a protein synthesis factory, maintaining proteostasis and ER homeostasis. However, a variety of intrinsic and extrinsic perturbations, such as cancer, can disrupt the
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